Biologia y terapia de la Fibromialgia. El estress, el sistema de respuesta al estress y la fibromialgia
Un último trabajo del Dr. Manuel Martinez Lavín en la enfermeda de Fibromialgia. Podeis acceder a su versión española accediendo al servicio de traducción que teneis a la izquierda.
Manuel Martinez-Lavin
National Institute of Cardiology, Juan Badiano 1, 14080 Mexico City, Mexico
Arthritis Research & Therapy 2007, 9:216 doi:10.1186/ar2146
Publicado 6 Julio 2007
Abstract
Stress is a state of disharmony, or threatened homeostasis. A stressor could have a psychological origin or a biological origin. Societies have become more intricate with industrialization, and modern individuals try to adapt to the new defiance by forcing their stress response system. The main component of the stress response network is the autonomic nervous system. The present article reviews current knowledge on autonomic dysfunction in fibromyalgia. Sympathetic hyperactivity has been consistently described by diverse groups of investigators. Fibromyalgia is proposed to be a sympathetically maintained neuropathic pain syndrome, and genomic data support this contention. Autonomic dysfunction may also explain other fibromyalgia features not related to pain.
Fuente|artritis-research
Manuel Martinez-Lavin
National Institute of Cardiology, Juan Badiano 1, 14080 Mexico City, Mexico
Arthritis Research & Therapy 2007, 9:216 doi:10.1186/ar2146
Publicado 6 Julio 2007
Abstract
Stress is a state of disharmony, or threatened homeostasis. A stressor could have a psychological origin or a biological origin. Societies have become more intricate with industrialization, and modern individuals try to adapt to the new defiance by forcing their stress response system. The main component of the stress response network is the autonomic nervous system. The present article reviews current knowledge on autonomic dysfunction in fibromyalgia. Sympathetic hyperactivity has been consistently described by diverse groups of investigators. Fibromyalgia is proposed to be a sympathetically maintained neuropathic pain syndrome, and genomic data support this contention. Autonomic dysfunction may also explain other fibromyalgia features not related to pain.
Fuente|artritis-research
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